Thoracic Outlet Syndrome Surgery in Sarasota & Bradenton
Arm Pain, Swelling, or Clots in a Young, Active Person? The Answer May Be in the Outlet.
Thoracic outlet syndrome (TOS) occurs when nerves, arteries, or veins become compressed in the thoracic outlet — the space between the collarbone and the first rib. Compression in this narrow passageway can cause a range of symptoms depending on which structures are affected, from arm pain and numbness to blood clots and impaired circulation.
TOS has three forms: neurogenic (nerve compression, the most common), venous (subclavian vein compression, causing swelling and clotting), and arterial (subclavian artery compression, the least common but most serious). Vascular Wellness Institute surgeons manage arterial and venous TOS — the vascular forms of this condition.
Venous TOS (Paget-Schroetter Syndrome)
Venous TOS — also called Paget-Schroetter syndrome or effort thrombosis — typically occurs in young, active individuals and athletes who engage in repetitive overhead arm activity. Compression of the subclavian vein causes clotting (deep vein thrombosis) in the arm, presenting with sudden arm swelling, heaviness, and discoloration. Treatment involves catheter-directed thrombolysis to dissolve the clot, followed by first-rib resection to decompress the thoracic outlet and prevent recurrence.
Arterial TOS
Arterial TOS is caused by compression of the subclavian artery, often in the setting of a cervical rib (an extra rib above the first rib) or an abnormal first rib. Chronic compression can damage the artery wall, leading to aneurysm formation, clot formation, and embolization to the hand and fingers. Treatment involves first-rib resection — and, if the artery has been damaged, arterial reconstruction.
Thoracic Outlet Decompression
The surgical treatment for vascular TOS is thoracic outlet decompression — most commonly first-rib resection, performed through an incision in the axilla (armpit) or above the collarbone. Removing the first rib widens the thoracic outlet and relieves the compression responsible for vascular symptoms. Recovery typically involves a hospital stay of one to two days and several weeks of activity restrictions.